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Acknowledgements The graphical abstract in this manuscript was created using BioRender.com(Agreement number: RY28QGNNJN)
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Such alterations were also associated with inhibition of cytochrome c oxidase (CcOX) activity and microtubule network disassembly, which are considered hallmarks of nitric oxide (NO) toxicity, indicating that NO, rather than the depletion of GSH per se, is the primary mediator of cell damage
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