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Tumor necrosis factor (TNF)‐like factor 1 (TLA‐1) is a homotrimeric member of the TNF family that is activated either membrane‐bound on lamina propria or cleaved into soluble form by proteases, leading to excessive inflammation via activation of Th2 immune responses
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Mast cell-derived tryptase can also activate proteinase-activated receptors on smooth muscle cells, leading to their contraction
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