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These findings are supported by many studies reporting the anti-inflammatory action of cortistatin on macrophages, neutrophils, and microglia in other conditions through mechanisms that involve both sstr and GHSR and the regulation of multiple intracellular signals [15,27,60,61,62,63,64]

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Next, we used the pro-inflammatory cytokines tumor necrosis factor-α (TNF-α) and interleukin-6 (IL-6) as indicators to evaluate the anti-inflammatory response