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Thus, iron availability not only responds to hypoxia but also actively regulates the hypoxic response, highlighting the bidirectional link between iron metabolism and HIF signaling

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Key molecular features include anchoring of the F23 residue within the receptor transmembrane bundle, an intramolecular salt bridge between E14 and R17 that stabilizes the helical structure, and interaction of the C‑terminal P37 with the receptor extracellular domain (ECD)